Is Test Flu a real phenomenon? What causes Test Flu? Can bad gear cause Test Flu?
Let’s delve into these questions and explore the concept of Test Flu together.
There are numerous factors to consider when examining Test Flu. Factors such as the age and genetics of the user, the cycle/compounds being utilized, and the quantity/dosage all play a role in the development of Test Flu. For instance, let’s assume a 25-year-old male is using testosterone and experiencing symptoms of Test Flu.
The question of Test Flu can be challenging to answer definitively due to the many variables involved. Factors such as the solvent/carrier used, quality and sterilization of the product, and any remaining acidic residues from the manufacturing process can all contribute to an immune response. Additionally, individual sensitivity, injection sites, and the route of administration can also impact the likelihood of experiencing Test Flu.
While we have a general idea of what may be causing Test Flu, there is a more plausible explanation to consider. It is important to remember that each individual’s experience with Test Flu may vary, and there may not be a one-size-fits-all answer. However, the following explanation may shed some light on the topic.
Testosterone is a complex hormone that can trigger direct and indirect reactions through various metabolites and molecular pathways. Understanding the intricacies of how testosterone interacts with the body can provide valuable insights into the development of Test Flu.
In conclusion, Test Flu is a multifaceted phenomenon that requires careful consideration of individual factors and circumstances. By exploring the various aspects of Test Flu, we can gain a better understanding of this intriguing topic.
Most commonly known are the following
(Example) Testosterone can have 3 roles
Most commonly known are the following
1) Being testosterone as its primary function…
2) Converting into DHT (dihydrotestosterone: a more potent metabolite/androgen)..
3)Converting into estrogen
Now, in addition to the fluctuations in hormones caused by exogenous testosterone increasing E1/E2, Testosterone, DHT, or other endocrine and adrenal secretions reaching abnormally high levels, the body’s static system strives to find balance and homeostasis. However, there is a crucial factor that is often overlooked and not well-known among the general population.
What about the metabolite Etiocholanolone? Could this be the culprit behind the phenomenon known as test flu? This stealthy offender may be responsible for the symptoms experienced – accountable for the “phenomenon” known as “test-flu”?
Who even knows what this so-called “bad guy” is?
Etiocholanolone, the 5-beta-reduced isomer of androsterone, is apparently a major metabolite of testosterone and androstenedione in various mammalian species, including humans. It’s excreted in urine and doesn’t do much in terms of androgenic activity. It’s classified as a ketosteroid, which apparently causes fever, acts as a pyrogen, stimulates the immune system, and increases white blood cell count.
The real kicker is that the purgogenic effect of Etiocholanolone is all thanks to the release of interleukin-1 from leukocytes that are mobilized in response to its production or injection. Oh, and let’s not forget that it also has anticonvulsant activity and might play a role in seizure susceptibility.
But wait, there’s more! Men with male pattern baldness might have significantly higher levels of etiocholanolone, along with testosterone and androstenone, in their urine. So, if you’re losing your hair, blame it on the etiocholanolone. Who knew this “bad-guy” had so many tricks up its sleeve?
Pyrogenicity of Etiocholanolone and Interleukin-1 in New and Old World Monkeys
Abstract
Etiocholanolone (5beta-androstan-3alpha-ol-17-one; referred to as E) is a significant metabolite of testosterone and androstenedione in various mammalian species, including humans. E, along with other 5beta-reduced steroids, has been identified as a fever-inducing agent in humans. The pyrogenic effect of these steroids is attributed to the release of interleukin-1 (IL-1) from leukocytes mobilized in response to steroid injections.
Old World Monkeys, such as Rhesus monkeys (Macaca mulatta), metabolize androgens similarly to humans, with E being a normal metabolite. Conversely, New World Monkeys, like Squirrel monkeys (Saimiri sciureus), lack hepatic 5alpha- and 5beta-steroid reductases, leading to the excretion of androgens in an unaltered state, thus E is not produced.
This study aims to investigate whether Squirrel monkeys have lost the ability to respond to 17-ketosteroids like E. Adult male Rhesus and Squirrel monkeys were treated with E, and their rectal temperatures were monitored over a 24-hour period. Rhesus monkeys showed a temperature increase of up to 3 degrees Fahrenheit following E injection. In contrast, Squirrel monkeys did not exhibit any rise in rectal temperature, even with doses up to 250 times the effective human dose. However, both species responded to injected IL-1alpha with a significant increase in rectal temperature.
The results indicate that E is pyrogenic in Rhesus monkeys but not in Squirrel monkeys. This supports the hypothesis that injected E may trigger IL-1 release in Rhesus monkeys but not in Squirrel monkeys.
Recent studies have confirmed that men are indeed more susceptible to illness, including fevers and the flu, due to their testosterone levels. So, the next time your significant other accuses you of exaggerating your symptoms, educate them on the concept of the “man flu” and why men may experience more severe symptoms. Rest assured, the scientific and medical community acknowledges and supports the challenges men face when it comes to their health. Embrace your testosterone-fueled resilience, gentlemen! And remember, the “man flu” is nothing to fear.
When the flu-like feverish symptoms subside, the ketosteroids levels return to normal, bringing the system back into balance. This can happen within days or sometimes last for weeks. Once levels are reduced, whether through the body’s natural homeostasis or medications like Ibuprofen or Benadryl, the subject can resume their testosterone injections without any recurring issues during that cycle. The introduction of other androgens may change this, but for most users, the likelihood is minimal. Some users have reported feeling lethargic, but fear not! This can be easily countered with B-complex supplements, DHEA, or the addition of GHRP-6 and/or Hexarelin. Keep reading to find out why these additions can be a game-changer.
This phenomenon is attributed to AAS (androgens) and can result in a decrease in peak plasma corticosterone concentrations by potentially inhibiting HSD-11B or 11B-HSD enzymes, which are responsible for converting cortisone to active cortisol. This can lead to symptoms resembling adrenal fatigue through various mechanisms of action.
Individuals may experience these effects while using certain AAS, such as Trenbolone, drol, and some pro-hormones (MT1/SD), which can result in a range of aggressive side effects. These side effects are not only due to liver toxicity but also through indirect pathways that produce undesirable outcomes in some users.
To mitigate these effects, it may be beneficial to incorporate a low to moderate dose of GHRP-6 and/or Hexarelin. These peptides act in a similar manner and have been shown to increase the release of ACTH, which can improve serum cortisol levels and restore them to a healthy functional state. This can help prevent symptoms resembling adrenal fatigue.
Sincerely,
Team EP
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